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Registro completo
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Biblioteca (s) : |
INIA Las Brujas. |
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Fecha : |
21/02/2014 |
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Actualizado : |
26/07/2021 |
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Tipo de producción científica : |
Actividades de Difusión |
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Autor : |
INIA (INSTITUTO NACIONAL DE INVESTIGACIÓN AGROPECUARIA); INIA LAS BRUJAS; PROGRAMA HORTICULTURA |
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Afiliación : |
ESTACIÓN EXPERIMENTAL "WILSON FERREIRA ALDUNATE", INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; PROGRAMA HORTICULTURA, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay. |
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Título : |
Presentación de resultados experimentales en ajo y cebolla. Reunión de divulgación. |
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Fecha de publicación : |
2000 |
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Fuente / Imprenta : |
Canelones (Uruguay): INIA, 2000. |
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Páginas : |
63 p. |
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Serie : |
(INIA Serie Actividades de Difusión ; 223) |
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Idioma : |
Español |
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Contenido : |
CONTENIDO - Introducción Mejoramiento genético de cebolla -- Conservación de bulbos de poblaciones locales de cebolla -- Conservación de bulbos de variedades de cebolla de día medio -- Evaluación de cultivares de cebolla -- Evaluación de un sistema de pronóstico para el control de enfermedades foliares en diferentes cultivares de cebolla -- Curado y almacenamiento de cebolla en macrotúnel -- Validación de tecnología de secado de cebollas -- Efecto de diferentes fechas de plantación en ajo blanco -- Efecto de diferentes fechas de plantación y tamaño de dientes en ejo elefante -- Efecto de diferentes dosis de nitrógeno y población de plantas en ajo blanco I -- Efecto de diferentes dosis de nitrógeno y población de plantas en ajo blanco II -- Mejoramiento genético de ajo -- Condiciones agroclimáticas en INIA Las Brujas. |
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Thesagro : |
AJO; ALLIUM CEPA; CEBOLLA; CONTROL DE ENFERMEDADES; EVALUACION DE CULTIVARES; FITOMEJORAMIENTO. |
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Asunto categoría : |
F01 Cultivo |
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URL : |
http://www.ainfo.inia.uy/digital/bitstream/item/302/1/18429200808115143.pdf
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Marc : |
LEADER 01549nam a2200229 a 4500
001 1006789
005 2021-07-26
008 2000 bl uuuu u00u1 u #d
100 1 $aINIA (INSTITUTO NACIONAL DE INVESTIGACIÓN AGROPECUARIA)
245 $aPresentación de resultados experimentales en ajo y cebolla. Reunión de divulgación.
260 $aCanelones (Uruguay): INIA$c2000
300 $a63 p.
490 $a(INIA Serie Actividades de Difusión ; 223)
520 $aCONTENIDO - Introducción Mejoramiento genético de cebolla -- Conservación de bulbos de poblaciones locales de cebolla -- Conservación de bulbos de variedades de cebolla de día medio -- Evaluación de cultivares de cebolla -- Evaluación de un sistema de pronóstico para el control de enfermedades foliares en diferentes cultivares de cebolla -- Curado y almacenamiento de cebolla en macrotúnel -- Validación de tecnología de secado de cebollas -- Efecto de diferentes fechas de plantación en ajo blanco -- Efecto de diferentes fechas de plantación y tamaño de dientes en ejo elefante -- Efecto de diferentes dosis de nitrógeno y población de plantas en ajo blanco I -- Efecto de diferentes dosis de nitrógeno y población de plantas en ajo blanco II -- Mejoramiento genético de ajo -- Condiciones agroclimáticas en INIA Las Brujas.
650 $aAJO
650 $aALLIUM CEPA
650 $aCEBOLLA
650 $aCONTROL DE ENFERMEDADES
650 $aEVALUACION DE CULTIVARES
650 $aFITOMEJORAMIENTO
700 1 $aINIA LAS BRUJAS
700 1 $aPROGRAMA HORTICULTURA
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Registro original : |
INIA Las Brujas (LB) |
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 | Acceso al texto completo restringido a Biblioteca INIA La Estanzuela. Por información adicional contacte bib_le@inia.org.uy. |
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Registro completo
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Biblioteca (s) : |
INIA La Estanzuela. |
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Fecha actual : |
02/03/2017 |
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Actualizado : |
07/03/2017 |
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Autor : |
GARCIA, J.P; GIANNITTI, F.; FINNIE, J.W.; MANAVIS, J. .; BEINGESSER, J.; ADAMS ,V.; ROOD, J.I.; UZAL, F.A. |
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Afiliación : |
FEDERICO GIANNITTI, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay. |
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Título : |
Comparative Neuropathology of Ovine Enterotoxemia Produced by Clostridium perfringens Type D Wild-Type Strain CN1020 and Its Genetically Modified Derivatives. |
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Fecha de publicación : |
2015 |
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Fuente / Imprenta : |
Veterinary Pathology, 2015, v.52.n.6. p.1250-1253, 2015. |
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Idioma : |
Inglés |
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Contenido : |
Abstract
Clostridium perfringens type D causes enterotoxemia in sheep and goats. The disease is mediated by epsilon toxin (ETX), which affects the cerebrovascular endothelium, increasing vascular permeability and leading to cerebral edema. In the present study, we compared the distribution and severity of the cerebrovascular changes induced in lambs by C. perfringens type D strain CN1020, its isogenic etx null mutant, and the ETX-producing complemented mutant. We also applied histochemical and immunohistochemical markers to further characterize the brain lesions induced by ETX. Both ETX-producing strains induced extensive cerebrovascular damage that did not differ significantly between each other in nature, neuroanatomic distribution, or severity. By contrast, lambs inoculated with the etx mutant or sterile, nontoxic culture medium did not develop detectable brain lesions, confirming that the neuropathologic effects observed in these infections are dependent on ETX production. Lambs treated with the wild-type and complemented strains showed perivascular and mural vascular edema, as well as serum albumin extravasation, particularly severe in the cerebral white matter, midbrain, medulla oblongata, and cerebellum. Brains of animals inoculated with the ETX-producing strains showed decreased expression of glial fibrillary acidic protein and increased expression of aquaporin-4 in the end-feet processes of the astrocytes around blood vessels. Early axonal injury was demonstrated with anti?amyloid precursor protein immunohistochemistry. Perivascular accumulation of macrophages/microglia with intracytoplasmic albumin globules was also observed in these animals. This study demonstrates that ETX is responsible for the major cerebrovascular changes in C. perfringens type D?induced disease. MenosAbstract
Clostridium perfringens type D causes enterotoxemia in sheep and goats. The disease is mediated by epsilon toxin (ETX), which affects the cerebrovascular endothelium, increasing vascular permeability and leading to cerebral edema. In the present study, we compared the distribution and severity of the cerebrovascular changes induced in lambs by C. perfringens type D strain CN1020, its isogenic etx null mutant, and the ETX-producing complemented mutant. We also applied histochemical and immunohistochemical markers to further characterize the brain lesions induced by ETX. Both ETX-producing strains induced extensive cerebrovascular damage that did not differ significantly between each other in nature, neuroanatomic distribution, or severity. By contrast, lambs inoculated with the etx mutant or sterile, nontoxic culture medium did not develop detectable brain lesions, confirming that the neuropathologic effects observed in these infections are dependent on ETX production. Lambs treated with the wild-type and complemented strains showed perivascular and mural vascular edema, as well as serum albumin extravasation, particularly severe in the cerebral white matter, midbrain, medulla oblongata, and cerebellum. Brains of animals inoculated with the ETX-producing strains showed decreased expression of glial fibrillary acidic protein and increased expression of aquaporin-4 in the end-feet processes of the astrocytes around blood vessels. Early axonal injury was demonstrated wi... Presentar Todo |
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Thesagro : |
ENFERMEDADES DE LOS ANIMALES. |
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Asunto categoría : |
-- |
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Marc : |
LEADER 02512naa a2200217 a 4500
001 1056750
005 2017-03-07
008 2015 bl uuuu u00u1 u #d
100 1 $aGARCIA, J.P
245 $aComparative Neuropathology of Ovine Enterotoxemia Produced by Clostridium perfringens Type D Wild-Type Strain CN1020 and Its Genetically Modified Derivatives.$h[electronic resource]
260 $c2015
520 $aAbstract Clostridium perfringens type D causes enterotoxemia in sheep and goats. The disease is mediated by epsilon toxin (ETX), which affects the cerebrovascular endothelium, increasing vascular permeability and leading to cerebral edema. In the present study, we compared the distribution and severity of the cerebrovascular changes induced in lambs by C. perfringens type D strain CN1020, its isogenic etx null mutant, and the ETX-producing complemented mutant. We also applied histochemical and immunohistochemical markers to further characterize the brain lesions induced by ETX. Both ETX-producing strains induced extensive cerebrovascular damage that did not differ significantly between each other in nature, neuroanatomic distribution, or severity. By contrast, lambs inoculated with the etx mutant or sterile, nontoxic culture medium did not develop detectable brain lesions, confirming that the neuropathologic effects observed in these infections are dependent on ETX production. Lambs treated with the wild-type and complemented strains showed perivascular and mural vascular edema, as well as serum albumin extravasation, particularly severe in the cerebral white matter, midbrain, medulla oblongata, and cerebellum. Brains of animals inoculated with the ETX-producing strains showed decreased expression of glial fibrillary acidic protein and increased expression of aquaporin-4 in the end-feet processes of the astrocytes around blood vessels. Early axonal injury was demonstrated with anti?amyloid precursor protein immunohistochemistry. Perivascular accumulation of macrophages/microglia with intracytoplasmic albumin globules was also observed in these animals. This study demonstrates that ETX is responsible for the major cerebrovascular changes in C. perfringens type D?induced disease.
650 $aENFERMEDADES DE LOS ANIMALES
700 1 $aGIANNITTI, F.
700 1 $aFINNIE, J.W.
700 1 $aMANAVIS, J. .
700 1 $aBEINGESSER, J.
700 1 $aADAMS ,V.
700 1 $aROOD, J.I.
700 1 $aUZAL, F.A.
773 $tVeterinary Pathology, 2015$gv.52.n.6. p.1250-1253, 2015.
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